Well, I don't see anything on that site for sending in a deer frozen brain to get tested. I only saw what one needs to do in case of a suspected human death from prion's. And they state you need to send in a piece of frozen brain for that.
yep, and our fine federal friends were going to destroy that brain collection that we donated our loved ones brain for research, scared of what science will finally discover $$$
www.upi.com/Science_News/2005/03/24/NIH-may-destroy-human-brain-collection/84811111671758/www.upi.com/Mad-Cow-Linked-to-thousands-of-CJD-cases/47861072816318/www.upi.com/Science_News/2005/04/01/Groups-seek-to-save-NIH-brain-collection/72961112392131/www.upi.com/NIH-sends-mixed-signals-on-CJD-brains/25701112902231/THURSDAY, AUGUST 17, 2017
*** Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States revisited 2017 ***
creutzfeldt-jakob-disease.blogspot.com/2017/08/monitoring-occurrence-of-emerging-forms.htmlatypical and typical bse, atypical and typical scrapie, and cwd, has all now been linked to sporadic cjd. regardless what the feds say, it is what it is, science shows this...
PRION 2015 CONFERENCE
O.05: Transmission of prions to primates after extended silent incubation periods: Implications for BSE and scrapie risk assessment in human populations
Emmanuel Comoy, Jacqueline Mikol, Valerie Durand, Sophie Luccantoni, Evelyne Correia, Nathalie Lescoutra, Capucine Dehen, and Jean-Philippe Deslys Atomic Energy Commission; Fontenay-aux-Roses, France
Prion diseases (PD) are the unique neurodegenerative proteinopathies reputed to be transmissible under field conditions since decades. The transmission of Bovine Spongiform Encephalopathy (BSE) to humans evidenced that an animal PD might be zoonotic under appropriate conditions. Contrarily, in the absence of obvious (epidemiological or experimental) elements supporting a transmission or genetic predispositions, PD, like the other proteinopathies, are reputed to occur spontaneously (atpical animal prion strains, sporadic CJD summing 80% of human prion cases). Non-human primate models provided the first evidences supporting the transmissibiity of human prion strains and the zoonotic potential of BSE. Among them, cynomolgus macaques brought major information for BSE risk assessment for human health (Chen, 2014), according to their phylogenetic proximity to humans and extended lifetime. We used this model to assess the zoonotic potential of other animal PD from bovine, ovine and cervid origins even after very long silent incubation periods.
*** We recently observed the direct transmission of a natural classical scrapie isolate to macaque after a 10-year silent incubation period,
***with features similar to some reported for human cases of sporadic CJD, albeit requiring fourfold long incubation than BSE. Scrapie, as recently evoked in humanized mice (Cassard, 2014),
***is the third potentially zoonotic PD (with BSE and L-type BSE),
***thus questioning the origin of human sporadic cases.
We will present an updated panorama of our different transmission studies and discuss the implications of such extended incubation periods on risk assessment of animal PD for human health.
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***thus questioning the origin of human sporadic cases***
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***our findings suggest that possible transmission risk of H-type BSE to sheep and human. Bioassay will be required to determine whether the PMCA products are infectious to these animals.
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prion2015.files.wordpress.com/2015/05/prion2015abstracts.pdfPRION 2016 CONFERENCE
Transmission data also revealed that several scrapie prions propagate in HuPrP-Tg mice with efficiency comparable to that of cattle BSE. While the efficiency of transmission at primary passage was low, subsequent passages resulted in a highly virulent prion disease in both Met129 and Val129 mice.
Transmission of the different scrapie isolates in these mice leads to the emergence of prion strain phenotypes that showed similar characteristics to those displayed by MM1 or VV2 sCJD prion.
These results demonstrate that scrapie prions have a zoonotic potential and raise new questions about the possible link between animal and human prions.
www.tandfonline.com/doi/abs/10.1080/19336896.2016.1163048?journalCode=kprn20www.tandfonline.com/doi/abs/10.1080/19336896.2016.1163048?journalCode=kprn20SPONTANEOUS ATYPICAL BOVINE SPONGIFORM ENCEPHALOPATHY
***Moreover, sporadic disease has never been observed in breeding colonies or primate research laboratories, most notably among hundreds of animals over several decades of study at the National Institutes of Health25, and in nearly twenty older animals continuously housed in our own facility.***
www.nature.com/articles/srep11573ELEPHANT IN THE ROOM $$$
LOOKING FOR CWD IN HUMANS AS nvCJD or as an ATYPICAL CJD, LOOKING IN ALL THE WRONG PLACES $$$
*** These results would seem to suggest that CWD does indeed have zoonotic potential, at least as judged by the compatibility of CWD prions and their human PrPC target. Furthermore, extrapolation from this simple in vitro assay suggests that if zoonotic CWD occurred, it would most likely effect those of the PRNP codon 129-MM genotype and that the PrPres type would be similar to that found in the most common subtype of sCJD (MM1).***
www.landesbioscience.com/journals/prion/article/28124/?nocache=112223249www.tandfonline.com/doi/full/10.4161/pri.28124?src=recsyswww.tandfonline.com/doi/pdf/10.4161/pri.28124?needAccess=trueTHURSDAY, AUGUST 17, 2017
JAVMA NEWS Atypical BSE found in Alabama cow September 01, 2017
transmissiblespongiformencephalopathy.blogspot.com/2017/08/javma-news-atypical-bse-found-in.htmlTHURSDAY, JULY 20, 2017
USDA OIE Alabama Atypical L-type BASE Bovine Spongiform Encephalopathy BSE animal feeds for ruminants rule, 21 CFR 589.200
bovineprp.blogspot.com/2017/07/usda-oie-alabama-atypical-l-type-base.htmlSUNDAY, JULY 23, 2017
atypical L-type BASE Bovine Amyloidotic Spongiform Encephalopathy BSE TSE PRION
bse-atypical.blogspot.com/2017/07/atypical-l-type-base-bovine-amyloidotic.htmlSUNDAY, JULY 23, 2017
Experimental Infection of Cattle With a Novel Prion Derived From Atypical H-Type Bovine Spongiform Encephalopathy
bse-atypical.blogspot.com/2017/07/experimental-infection-of-cattle-with.htmlWEDNESDAY, JULY 26, 2017
APHIS USDA Emerging Animal Disease Preparedness and Response Plan July 2017
animalhealthreportpriontse.blogspot.com/2017/07/aphis-usda-emerging-animal-disease.htmlTHURSDAY, JULY 13, 2017
EFSA BSE Sixty cases of mad cow disease since 2001 breached feed ban likely the cause
Scientists investigate origin of isolated BSE cases
bovineprp.blogspot.com/2017/07/efsa-bse-sixty-cases-of-mad-cow-disease.htmlTUESDAY, AUGUST 8, 2017
Concurrence With OIE Risk Designations for Bovine Spongiform Encephalopathy [Docket No. APHIS-2016-0092]
animalhealthreportpriontse.blogspot.com/2017/08/concurrence-with-oie-risk-designations.htmlkind regards, terry
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