Just read a few studies that say CWD prions remain infectious after passage through the digestive system of coyotes and crows. We all know crows eat eyeballs libs and butt holes and Coyotes eat gut piles. So the prions are more then likely spread around. If their feces fertilizes a plant and the deer then eats that plant then the deer gets CWD.
Is this fact or is it fiction?
i was just going over this old data...see;
-----Original Message-----
From: Terry Singeltary <flounder9@verizon.net>
To: flounder9 <flounder9@verizon.net>
Sent: Sat, Sep 21, 2019 10:09 am
Subject: scavengers and cwd
It has been reported (8) the ability of chronic wasting disease (CWD) -infected brain material to pass through the gastrointestinal tract of coyotes (Canis latrans) following oral ingestion, and be infectious, demonstrating that mammalian scavengers could contribute to the translocation and contamination of CWD in the environment.
CWD prions remain infectious after passage through the digestive system of coyotes (Canis latrans)
Tracy A Nichols,Justin W Fischer,Terry R Spraker,Qingzhong Kong &Kurt C VerCauteren
Pages 367-375 | Received 07 Jul 2015, Accepted 18 Aug 2015, Accepted author version posted online: 04 Dec 2015, Published online: 21 Dec 2015
www.tandfonline.com/doi/full/10.1080/19336896.2015.1086061American crows (Corvus brachyrhynchos) and potential spreading of CWD through feces of digested infectious carcases
Could Crows Play a Role in Spreading CWD
was presented by Dr. Kurt VerCauteren, NWRC, WS-APHIS- USDA. From the first observations (40 years ago) of CWD in mule deer (Odocoileus hemionus) and Rocky Mountain elk (Cervus elaphus nelsoni) in Northern Colorado, the disease has been identified in an increasing geographic area. Mechanisms for the spread of CWD are incompletely understood. Birds have been identified as potential vectors for a number of diseases, where infected material is ingested and the disease agent is later shed in new areas after flying substantial distances. We hypothesized that avian scavengers have the potential to disseminate 200 prions associated with transmissible spongiform encephalopathies (TSEs), like CWD, by a similar process. As prions are resistant to destruction, it is reasonable that infectious material could pass through the digestive tract of scavenging birds. Our objective was to determine if TSE-positive brain material from mice (i.e., mouse-adapted scrapie) could pass through the digestive tract of American crows (Corvus
American crows (Corvus brachyrhynchos) and potential spreading of CWD through feces of digested infectious carcasses
brachyrhynchos) and still be infectious to mice. Our experimental design included treatment groups of mice inoculated intraperitoneally with: 1) normal mouse brain, 2) infected mouse brain, 3) gamma-irradiated feces from crows gavaged with normal mouse brain, and 4) gamma-irradiated feces from crows gavaged with infected mouse brain. Our preliminary results indicate feces from each of 20 crows gavaged with infected mouse brain were infectious for mice (proportion of crows=1.00, 95% CI: 0.83-1.00) and average longevity for mice was 213 days (95% CI: 210-216). Longevity of mice inoculated with infected mouse brain was slightly less (198 days, 95% CI: 188-207). Most mice inoculated with normal brain, or feces from crows gavaged with normal brain, were still alive 1 year post inoculation with no evident clinical signs of TSE disease in any control mice. Our results demonstrate that a common, migratory North American scavenger, the American crow, can pass infective prions in feces and, therefore, could play a role in the spatial dissemination of prion disease....
Chronic wasting disease (CWD), first identified in Wisconsin in 2002, is an infectious transmissible spongiform encephalopathy (TSE) afflicting members of the taxonomic family Cervidae, and causes neurodegeneration and ultimately death. As a proxy for mortality or harvest of CWD-infected deer, we placed disease-free white-tailed deer (Odocoileus virginianus) carcasses and gut piles in the environment and monitored scavenger activity and carcass removal from September to April in 2003 through 2005. We recorded 14 species of scavenging mammals (six species of visitors), and eight species of scavenging birds (14 species of visitors). Prominent scavengers included American crows (Corvus brachyrhynchos), raccoons (Procyon lotor), and Virginia opossums (Didelphis virginiana). We found no evidence that deer directly consumed conspecific remains, but they visited carcasses and gut piles. Domestic dogs (Canis familiaris), cats (Felis sylvestris catus), and cows (Bos spp.) either scavenged or visited carcass sites, which may have increased exposure risk of CWD to humans and human food supplies. Deer carcasses persisted for a median of 18 to 101 days, while gut piles lasted for a median of three days. Habitat did not influence carcass consumption/decomposition, but mammalian and avian scavenger activity and higher temperatures (proxy for microbial and arthropod activity) were associated with greater rates of carcass removal. Our findings suggest that infected deer carcasses can function as an environmental source of CWD prions to mammalian and avian scavengers. We discuss the implications of these results in a broader context of CWD spread, and suggest preemptive management strategies for mitigating impacts of CWD contaminated deer remains in the environment....
www.usaha.org/meetings/2008/ LINK IS DEAD
THIS potential vector of transmission is very disturbing. I don't know what the flight paths, and or travel of either species of birds, or if there are others, there are many birds that eat meat and or are scavengers. But the disturbing part is the amount of territory they can cover and spread their feces. PLUS, this goes back to what the late Dr. Gibbs told me, and what the late Harash Narang book showed, Dr. Gibbs stating that the TSE agent could spread through the digestinal track, and survive, and could still have the potential to spread, and Harash Narang's book 'The Link', page 135, where a farmers around Kent have chickens with BSE. MAFF was aware of this and was suppose to do some studies? BUT, regardless whether or not these birds become clinical and die, the fact that the above studies showed that the TSE agent survived the digestinal tract, and went on to further infect mice via feces, is very disturbing, and further enhances transmission studies must be done asap. PLUS, this should be the final straw for chicken litter being fed back to cattle and other food producing animals for humans and animals. AND not to forget the Red Necked Ostrich and BSE? ...TSS
FRIDAY, DECEMBER 06, 2013
Procedures for identifying infectious prions after passage through the digestive system of an avian species
chronic-wasting-disease.blogspot.com/2013/12/procedures-for-identifying-infectious.htmlTHURSDAY, MARCH 08, 2018
Cervid, Wild Hogs, Coyotes, Wolves, Cats, Rodents, Gut Piles and Scavengers, A Potential Risk as Regards Disease Transmission CWD TSE Prion
chronic-wasting-disease.blogspot.com/2018/03/cervid-wild-hogs-coyotes-wolves-cats.htmlTHURSDAY, AUGUST 08, 2019
Raccoons accumulate PrPSc after intracranial inoculation with the agents of chronic wasting disease (CWD) or transmissible mink encephalopathy (TME) but not atypical scrapie
chronic-wasting-disease.blogspot.com/2019/08/raccoons-accumulate-prpsc-after.htmlWEDNESDAY, JULY 31, 2019
The agent of transmissible mink encephalopathy passaged in sheep is similar to BSE-L
transmissible-mink-encephalopathy.blogspot.com/2019/07/the-agent-of-transmissible-mink.htmlFELINE SPONGIFORM ENCEPHALOPATHY FSE AND CANINE SPONGIFORM ENCELPHALOPATHY CSE
felinespongiformencephalopathyfse.blogspot.com/caninespongiformencephalopathy.blogspot.com/MONDAY, FEBRUARY 25, 2019
MAD DOGS AND ENGLISHMEN BSE, SCRAPIE, CWD, CJD, TSE PRION A REVIEW 2019
bseinquiry.blogspot.com/2019/02/mad-dogs-and-englishmen-bse-scrapie-cwd.htmlnow, here is what is so bothersome about this whole mess, and read this very carefully.
the mad cow feed ban was a farce. it is still legal to feed cows and everything else, including deer and elk, cervid that have been rendered into feed, without testing for cwd tse prion. WE NOW KNOW THAT CWD AND SCRAPIE WILL TRANSMIT TO PIGS BY ORAL ROUTES, plus we have another outbreak of tse prion in a new livestock species i.e. the camel in Africa, and it's rather large from what i am hearing. all this adds up to one thing, very bad news...they have just admitted that the mad cow feed ban never really existed...see;
SUNDAY, SEPTEMBER 1, 2019
FDA Reports on VFD Compliance
bovineprp.blogspot.com/2019/09/fda-reports-on-vfd-compliance.htmlTHURSDAY, SEPTEMBER 26, 2019
Veterinary Biologics Guideline 3.32E: Guideline for minimising the risk of introducing transmissible spongiform encephalopathy prions and other infectious agents through veterinary biologics
bovineprp.blogspot.com/2019/09/veterinary-biologics-guideline-332e.htmlSATURDAY, SEPTEMBER 28, 2019
Texas CWD TSE Prion aka Mad Deer Disease Detected Free Range Mule Deer El Paso 145 Positive To Date
chronic-wasting-disease.blogspot.com/2019/09/texas-cwd-tse-prion-aka-mad-deer.htmlhay, straw, grains...and cwd tse prion
***> NORWAY CWD UPDATE December 2018 Report from the Norwegian Scientific Committee for Food and Environment (VKM) 2018: 16
Factors that can contribute to spread of CWD – an update on the situation in Nordfjella, Norway
Opinion of Panel on biological hazards of the Norwegian Scientific Committee for Food and Environment 13.12.2018 ISBN: 978-82-8259-316-8 ISSN: 2535-4019 Norwegian Scientific Committee for Food and Environment (VKM) Po 222 Skøyen 0213 Oslo Norway FRIDAY, DECEMBER 14, 2018 Norway, Nordfjella VKM 2018 16
Factors that can contribute to spread of CWD TSE Prion UPDATE December 14, 2018
vkm.no/download/18.696229a71677d983532c0c11/1544792739325/CWD%20factors%20for%20spread%202018.pdf chronic-wasting-disease.blogspot.com/2018/12/norway-nordfjella-vkm-2018-16-factors.html***>>> Recently, we have been using PMCA to study the role of environmental prion contamination on the horizontal spreading of TSEs. These experiments have focused on the study of the interaction of prions with plants and environmentally relevant surfaces. Our results show that plants (both leaves and roots) bind tightly to prions present in brain extracts and excreta (urine and feces) and retain even small quantities of PrPSc for long periods of time. Strikingly, ingestion of prioncontaminated leaves and roots produced disease with a 100% attack rate and an incubation period not substantially longer than feeding animals directly with scrapie brain homogenate. Furthermore, plants can uptake prions from contaminated soil and transport them to different parts of the plant tissue (stem and leaves). Similarly, prions bind tightly to a variety of environmentally relevant surfaces, including stones, wood, metals, plastic, glass, cement, etc. Prion contaminated surfaces efficiently transmit prion disease when these materials were directly injected into the brain of animals and strikingly when the contaminated surfaces were just placed in the animal cage. These findings demonstrate that environmental materials can efficiently bind infectious prions and act as carriers of infectivity, suggesting that they may play an important role in the horizontal transmission of the disease.
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Since its invention 13 years ago, PMCA has helped to answer fundamental questions of prion propagation and has broad applications in research areas including the food industry, blood bank safety and human and veterinary disease diagnosis.
prion2015.files.wordpress.com/2015/05/programguide1.pdf THURSDAY, OCTOBER 25, 2018
***> Norway New additional requirements for imports of hay and straw for animal feed from countries outside the EEA due to CWD TSE Prion
www.mattilsynet.no/dyr_og_dyrehold/for/nye_regler_om_tilleggskrav_ved_import_av_hoy_og_halm_til_dyrefor.32299 chronic-wasting-disease.blogspot.com/2018/10/norway-new-additional-requirements-for.htmlkind regards, terry
